Skip Navigation
Skip to contents

ICDM 2019

Main symposia

S2 IR and inflammation
Insulin resistance and systemic inflammation: 'Axis of evil' in diabetes
15:30~17:30 / Thursday 10 October
Emerald hall B
Chairman:Ki-Up Lee, Carol H. Wysham
Overview

Organizers: Shin Ae Kang

This session titled, 'Insulin resistance and systemic inflammation: 'Axis of evil' in diabetes' is a basic session designed for both basic and clinical science researchers. It will present the mechanism as well as the consequences of insulin resistance. The talks will be focused on demonstrating how inflammation, including the immune cells and its cytokines, contributes to the development of glucose intolerance and atherosclerosis and how hyperinsulinemia may perpetuate inflammation and various metabolic, non-metabolic consequences. The session will provide a new understanding of the cross-talk between the insulin resistance and inflammation that will help us overcome this 'axis of evil' in the future.
Goo Taeg Oh
Ewha Womans University, Korea
2-1Vascular and immune cell network in the pathogenesis of atherosclerosis
James D. Johnson
University of British Columbia, Canada
2-2Causal effects of hyperinsulinemia on insulin resistance, obesity and aging
Seung-Soon Im
Keimyung University, Korea
2-3SREBP-1a-stimulated lipid synthesis is required for macrophage phagocytosis
Minho Shong
Chungnam National University, Korea
2-4Interactive roles of Th2 cytokines and GDF15 in glucose homeostasis
S5 Islet biology
Intracellular environment for beta cell function
09:00~10:40 / Friday 11 October
Emerald hall B
Chairman:Kwang-Won Kim, James Johnson
Overview

Organizers: Hail Kim

This session titled, 'Intracellular environment for beta cell function' is a basic session that is designed to provide an opportunity for clinicians and basic researchers to understand the importance of intracellular environment for beta cell function and mass. In this session, a variety of factors affecting intracellular environment of cell will be discussed. Researchers will present the interaction between cellular environment and genes, autophagy and mitochondria in beta cells. These studies will provide a potential possibility of developing anti-diabetes drug targeting beta cells.
Yoshiaki Kido
Kobe University, Japan
5-1Regulation of pancreatic β cell mass from the interaction of gene-environmental factors
Myung-Shik Lee
Yonsei University, Korea
5-2An autophagy enhancer expedites clearance of islet amyloid and ameliorates human-type diabetes
Hyeongseok Kim
Chungnam National University, Korea
5-3Lactation improves glucose homeostasis by enhancing beta cell mass and function
Kun Ho Yoon
The Catholic University of Korea, Korea
5-4Targeting PGC-1 to improve glucolipotoxicity induced beta-cell dysfunction
S9 Central control of metabolism
Central regulators of energy metabolism
13:20~15:20 / Friday 11 October
Emerald hall B
Chairman:Jae Bum Kim, Graeme I. Bell

Organizer: Min Seon Kim

The hypothalamus plays a central role in maintain normal body weight and normal glucose levels. Emerging evidence suggests that communications between neurons and glial cells in the hypothalamus as well as crosstalk between the hypothalamus and peripheral organs are critical for energy balance. The first speaker, Dr. Min-Seon Kim from the Asan Medical Center, Korea, will present that hypothalamic neuronal mitochondrial stress differently affects fat tissue metabolism depending on the degree of stress. The second speaker, Dr. Jae-Geun Kim from Incheon University, Korea, will talk about the importance of hypothalamic neuron-microglial interactions in the control of energy metabolism. The last speaker, Dr. Suneil Koliwad from the UCSF, USA, will present the data indicating microglia as a fat sensitive mediator in obesity-associated metabolic disorders. Hopefully, this symposium will provide novel information about the mechanisms of central regulation of energy metabolism to the audience.
Min-Seon Kim
University of Ulsan, Korea
9-1Hypothalamic neuron mitohormesis and fat tissue metabolism
Jae Geun Kim
Incheon National University, Korea
9-2Hypothalamic neuron-glia interactions and energy metabolism
Suneil K. Koliwad
University of California San Francisco, USA
9-3Adaptive and maladaptive roles of hypothalamic microglia
C. Justin Lee
The Institute for Basic Science, Korea
 C. Justin Lee
9-4Role of hypothalamic reactive astrocytes in obesity
S14 Aging and metabolic disease
Aging, the inevitable player in the development of metabolic disease
16:20~18:00 / Friday 11 October
Emerald hall A
Chairman:Cheol Soo Choi, Florin Despa
Overview

Organizers: Shin Ae Kang

This session titled, 'Aging, the inevitable player in the development of metabolic disease' is a translational session that is designed to provide an in-depth discussion for basic as well as translational science researchers. It will help an audience understand novel mechanisms underlying aging-associated metabolic dysfunction. The talks will be focused on demonstrating novel molecular and cellular mechanisms that link aging and various metabolic diseases together with possible therapeutic options that may help alleviate aging-associated metabolic dysfunction. The session will provide a new understanding of aging in the aspect of the metabolic disease and the potential future therapeutic targets.
Yong Ryoul Yang
Korea Research Institute of Bioscience & Biotechnology, Korea
14-1A novel myokine (Myo-X) induces skeletal muscle hypertrophy and reverses age-related muscle weakness
Jae Myoung Suh
KAIST, Korea
14-2Mapping the mitochondrial proteome in aging and disease
Chanhee Kang
Seoul National University, Korea
14-3Cellular senescence and its associated metabolites
Eo Su Kim
Yonsei University, Korea
14-4AMPK in Alzheimer's disease: friend or foe?
r
S15 NAFLD
Evolving views on the pathophysiology of NAFLD
16:20~18:00 / Friday 11 October
Emerald hall B
Chairman:Myung-Shik Lee, Suneil K. Koliwad
Overview

Organizers: Hye Soon Kim, Dae Ho Lee

This session titled, 'Evolving views on the pathophysiology of NAFLD' is a translational session that is designed to provide an opportunity for clinicians and basic researchers to understand novel pathophysiologies and potential therapeutic areas for NAFLD. In this session, a variety of therapeutic targets based on the novel NAFLD pathophysiologies, including biological network models to integrate multiomics data, autophagy, NAD+ metabolism, and important immunologic regulators, will be presented and discussed. This session will be the first step toward a better understanding the importance of NAFLD in T2D.
Adil Mardinoglu
King's College London, UK
15-1The use of systems biology in treatment of liver diseases
Young Mi Jung
Pusan National University, Korea
15-2Therapeutic potential of TSG-6 for treating liver disease
Dongryeol Ryu
Sungkyunkwan University, Korea
15-3NAD+ metabolism and liver disease
S19 SGLT2 inhibitor
SGLT2 inhibitor, beyond glucosuric effect
09:00~11:00 / Saturday 12 October
Emerald hall A
Chairman:Hanyang University, Korea, Stefano Del Prato
Overview

Organizers: Yong Ho Lee

This session entitled with 'SGLT2 inhibitor, beyond glucosuric effect' is basic and translational session and designed to provide a place for basic researchers and clinicians to understand the novel non-hypoglycemic effects of SGLT2 inhibitors on metabolism and physiology. In this session, various actions and mechanisms of SGLT2 inhibitors regarding kidney protection, insulin secretory function of beta cells and ketone body metabolism will be presented and discussed by outstanding international speakers. It will also cover the overview of SGLT2 inhibitors to link between physiology and clinical outcomes from cardiovascular safety trials.
Roger Chen
University of Sydney, Australia
19-1SGLT2 inhibitors in metabolism- from physiology to clinical outcomes
Takayuki Miki
Sapporo University, Japan
19-2SGLT2 inhibitors reduced acute kidney injury and mortality after myocardial infarction by inhibition of oxidative stress in diabetic rats
Young Min Cho
Seoul National University, Korea
19-3Sodium-glucose cotransporter-2 inhibition and beta-cell function
Yong Ho Lee
Yonsei University, Korea
19-4SGLT2i regulates ketone body metabolism via interorgan crosstalk
S20 Browning and beiging
Metabolic implication of browning and whitening
09:00~11:00 / Saturday 12 October
Emerald hall B
Chairman:Eulji University, Korea, Adil Mardinoglu
Overview

Organizers: Eun Hee Koh

This session titled, 'Metabolic implication of browning and whitening' is a basic session that is designed to provide novel insights into the pathophysiology of obesity. In this session, metabolic reprogramming of adipose tissue in the obesity and environmental stress and the physical characteristics of adipose tissue will be presented and discussed. This session will be a great opportunity to better understand the novel target of obesity in the metabolic adaptation of adipose tissue.
Shingo Kajimura
University of California, USA
20-1Metabolic adaptation in physiology and disease
Lei Sun
National University of Singapore, Singapore
20-2Adipose circular RNAs exhibit dynamic regulation in obesity and functional role in adipogenesis
Seung-Hoi Koo
Korea University, Korea
20-3Role of adipose tissues in energy homeostasis
Yun-Hee Lee
Seoul National University, Korea
20-4Single cell transcriptomic profiling of adipocyte progenitors characterized molecular basis of beige adipogenesis
  • Korean Diabetes Association
    101-2104, Lotte Castle President, 109
    Mapo-gu, Seoul, 04146, Korea

    E-mail : icdm@diabetes.or.kr

  • Congress Secretariat (The Within)
    5F, Samgyeong Bldg., #100-20, Yanghwa-ro, Mapo-gu, Seoul, Korea

    TEL : 82-2-6959-5333 FAX : . 82-70-8677-6333 E-mail : icdm@diabetes.or.kr